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In AD, disruption in FOCM accelerates amyloid- deposition, tau phosphorylation, cognitive decline, and vascular compromise [113, 114, 116, 117] which can be mitigated by folic acid supplementation in experimental studies [115], and is consistent with clinical evidence showing low folate and elevated homocysteine in patients [6, 8], though supplementation trials remain inconclusive [104, 110]

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147 In an intrahepatic cholestasis of pregnancy rat model, 17-ethinylestradiol treatment leads to reduced gut flora -diversity and significantly altered structure

2023 Mar 3;12(1):6