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Tesaglitazar-induced cardiac dysfunction was associated with accumulation of cardiac lipids, including lipids that have been linked with cardiac lipotoxicity, such as acyl-carnitines and diacylglycerols (11, 50)

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The researchers showed that senescent Leydig cell accumulation is a driver of age-related testosterone decline, and that targeted elimination of these cells with FOXO4-DRI restored endocrine function

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