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Several transcriptional factors are involved in this process, such as sterol regulatory element-binding transcription factor-1c (SREBF-1c) [77] and peroxisome proliferator-activated receptor (PPAR) [78], which can be regulated by non-coding RNAs (e.g., miR-615-5p and miR-130a)

276 In summary, bronchiectasis is a disease fueled by a vicious cycle of continuous bacterial infections and a process of environmental dysregulation, accompanied by tissue injury and damaged lung function

Eicosanoid production by different cell types within the tumor microenvironment further complicates the profile tumor-associated macrophages contribute PGE2 and LTB4, while the tumor cells themselves express varying profiles of COX-2 and downstream prostaglandin synthases depending on the tumor type