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Keywords : copper, cuproptosis, copper homeostasis, cardiovascular disease, mitochondrial dysfunction Cell death is a critical driver of cardiovascular disease pathogenesis, contributing to tissue damage, dysfunction, and inflammation [1]

Human clinical studies report measurable reductions in HbA1c levels, body-weightrelated endpoints, and improved insulin sensitivity markers compared to placebo-controlled groups

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Due to the influx of sick patients, hospital area pharmacies usually have a stronger medical supply