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As MOTS-c activates AMPK and NRF2, it also indirectly suppresses the mTOR (mechanistic target of rapamycin) pathway, the master growth and anabolism regulator that, when chronically overactivated, accelerates the accumulation of senescent cells

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doi: 10.1016/j.jaut.2011.01.002 37 KallmannBAHtherMTubesMFeldkampJBertramsJGriesFAet al

Research-grade GHK-Cu is produced by solid-phase peptide synthesis of the Gly-His-Lys tripeptide using standard Fmoc protecting-group chemistry, followed by copper complexation