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doi: 10.1007/s00253-021-11558-7

Together, these observations reveal an intramolecular handover mechanism for the formation of the :2 hemicomplex

FPN caused hippocampal microglia and astrocytes to proliferate and become more active, potentially increasing inflammatory cytokine expression 14 , such as IL-6, TNF- , and IL-1 , which could exacerbate oxidative stress and inflammation 3